Abstract

Osteomyelitis is a progressive inflammation of the bone as well as bone marrow, and also in the facial skeleton, it is most commonly seen in the tooth bearing area [1]. The incidence of osteomyelitis higher in the mandible owing to the dense poorly vascularized cortical plates and the blood supply primarily from the inferior alveolar neurovascular bundle. Poor host defences and infective foci, both local and systemic, can contribute significantly to the emergence and clinical course of the disease [2]. Various classification systems for osteomyelitis have been documented in literature, such as suppurative or nonsuppurative, hematogenous or secondary to a contiguous focus of infection, and acute or chronic, with the latter becoming the predominant classification system [1]. Osteomyelitis can also be acute and chronic forms. Acute osteomyelitis can be further subdivided into suppurative and nonsuppurative forms as well as progressive or hematogenous forms. Chronic osteomyelitis may be classified by the causative agent or as suppurative or nonsuppurative forms or sclerosing with subclassifications of diffuse or focal disease [1].